Supplementing their diet with glucosamine, people with arthritis may slow their bone turnover and help build cartilage, according to a new animal study published in Arthritis and Rheumatism.
The study by Suanne Wane and colleagues from the University of Toronto in Ontario, Canada examined a building block of proteins known as glycosaminoglycans, which the body uses to build cartilage, in rabbits with arthritis.
In the study, rabbits were given 100 mg of glucosamine a day for eight weeks and the researchers compared two groups of rabbits with arthritis, one given glucosamine and the other given a placebo for the bone turnover rates.
Wang and team found rabbits supplemented with glucosamine had a bone turnover rate around the knee joint similar to that of normal animals while untreated rabbits had a higher rate.
Saturday, June 30, 2007
Sunday, June 17, 2007
Thursday, June 14, 2007
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Thursday, June 07, 2007
Sugar supplement hope for diabetes
Scientists discovered that a compound called N-acetyl glucosamine (GlcNAc) prevented the development of diseases equivalent to MS and type 1 diabetes in tests on mice.
A team from the University of California, Irvine found GlcNAc molecules suppressed damaging autoimmune responses, New Scientist reported.
They do this by attaching to receptors on the outside of immune cells, preventing them from attacking the body's own cells and tissue, the researchers discovered.
Experiments in 2002 showed that glucosamine - which is similar to GlcNAc but less potent - worked as well as regular immunosuppressants in increasing the amount of time transplanted hearts lasted in mice.
Nick Giannoukakis, a pathologist at the University of Pittsburgh School of Medicine in Pennsylvania, who carried out the earlier study, said he was "astounded" by the latest results.
But he warned that further work was needed to show that glucosamine or GlcNAc could reverse symptoms in animals with autoimmune diseases as well as prevent them.
There is already some evidence to support this - in 2005 researchers revealed that glucosamine could suppress MS symptoms in mice that had recently developed the disease.
A team from the University of California, Irvine found GlcNAc molecules suppressed damaging autoimmune responses, New Scientist reported.
They do this by attaching to receptors on the outside of immune cells, preventing them from attacking the body's own cells and tissue, the researchers discovered.
Experiments in 2002 showed that glucosamine - which is similar to GlcNAc but less potent - worked as well as regular immunosuppressants in increasing the amount of time transplanted hearts lasted in mice.
Nick Giannoukakis, a pathologist at the University of Pittsburgh School of Medicine in Pennsylvania, who carried out the earlier study, said he was "astounded" by the latest results.
But he warned that further work was needed to show that glucosamine or GlcNAc could reverse symptoms in animals with autoimmune diseases as well as prevent them.
There is already some evidence to support this - in 2005 researchers revealed that glucosamine could suppress MS symptoms in mice that had recently developed the disease.
Tuesday, June 05, 2007
Glucosamine-like supplement inhibits multiple sclerosis, type 1 diabetes
(NewsTarget) A glucosamine-like dietary supplement has been found to suppress the damaging autoimmune response seen in multiple sclerosis and type 1 diabetes mellitus, according to University of California, Irvine health sciences researchers.
In studies on mice, Dr. Michael Demetriou and colleagues with the UC Irvine Center for Immunology found that N-acetylglucosamine (GlcNAc), which is similar but more effective than the widely available glucosamine, inhibited the growth and function of abnormal T-cells that incorrectly direct the immune system to attack specific tissues in the body, such as brain myelin in MS and insulin-producing cells of the pancreas in diabetes. Study results appear on the online version of the Journal of Biological Chemistry.
"This finding shows the potential of using a dietary supplement to help treat autoimmune diseases," said Demetriou, an assistant professor of neurology, and microbiology and molecular genetics. "Most importantly, we understand how this sugar-based supplement inhibits the cells that attack the body, making metabolic therapy a rational approach to prevent or treat these debilitating diseases."
In studies on mice, Dr. Michael Demetriou and colleagues with the UC Irvine Center for Immunology found that N-acetylglucosamine (GlcNAc), which is similar but more effective than the widely available glucosamine, inhibited the growth and function of abnormal T-cells that incorrectly direct the immune system to attack specific tissues in the body, such as brain myelin in MS and insulin-producing cells of the pancreas in diabetes. Study results appear on the online version of the Journal of Biological Chemistry.
"This finding shows the potential of using a dietary supplement to help treat autoimmune diseases," said Demetriou, an assistant professor of neurology, and microbiology and molecular genetics. "Most importantly, we understand how this sugar-based supplement inhibits the cells that attack the body, making metabolic therapy a rational approach to prevent or treat these debilitating diseases."
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